Magnesium - Mg
In the body it works closely with calcium and phosphorus — about 60% of magnesium is found in bones and teeth. While calcium is responsible for muscle contraction, magnesium enables their proper relaxation. It is a cofactor for hundreds of enzymatic reactions, supports nerve conduction, and plays a key role in energy metabolism at the cellular level (ATP production) [1]. The NRC standard for an adult dog is about 150 mg / 1000 kcal ME (minimum 45 mg) [1].
Where it's found: Red meat (beef, game, lamb), hearts (especially beef and lamb), kidneys, and fish. Raw meaty bones and cartilage tissue provide a certain amount, but their excess paradoxically lowers magnesium absorption.
Deficiency: Resulting directly from too little magnesium in a meat-based diet is rare; most often it is a secondary effect of chronic diarrhea or kidney disease. In the calculator, a magnesium deficiency with correct calcium and phosphorus almost always means that the diet has too little red meat and hearts relative to bone — this is a better "sensor" of recipe quality than the Ca:P ratio alone [2]. The solution is correcting the composition, not supplementation.
Excess: In animals with properly functioning kidneys, dietary excess does not occur — the body efficiently excretes magnesium in urine [1]. Excess is practically possible only from supplements: it then causes diarrhea, and in extreme cases lethargy.
Species difference — urine and stones
In cats, magnesium stirs more emotion than in dogs because of its role in the formation of urinary stones. The historical "magnesium = struvite" panic, however, concerns mainly dry foods — a raw meat diet naturally acidifies the urine, which drastically reduces this risk [3]. What's more, overly aggressive lowering of magnesium in a cat with acidic urine may paradoxically favor oxalate stones instead of struvite ones [4]. Don't cut magnesium by force — hydration, pH, and overall urine balance are more important; a check-up urine test 1–2 times a year is good practice regardless of diet.
What to watch for
Excess calcium from bone competes with magnesium for the same transporters in the gut (TRPM6/TRPM7), so a diet with a lot of bone (above 15–20%) can lead to a functional magnesium deficiency despite its presence in the bones themselves [1]. More on the proportion in the article calcium:phosphorus ratio. Diuretics (e.g. furosemide) increase magnesium excretion — with ongoing pharmacotherapy or kidney disease, the magnesium balance is worth consulting with the attending veterinarian. If supplementation is necessary, glycinate is better tolerated than citrate or magnesium oxide.
Sources
- National Research Council (2006). Nutrient Requirements of Dogs and Cats, Chapter 7 — Minerals. National Academies Press. https://nap.nationalacademies.org/catalog/10668/nutrient-requirements-of-dogs-and-cats
- Hand MS, Thatcher CD, Remillard RL, Roudebush P, Novotny BJ (2010). Small Animal Clinical Nutrition, 5th Edition. https://www.markmorrisinstitute.org/sacn5.html
- Osborne CA, et al. (1984). Treatment and prevention of feline struvite urolithiasis. https://pubmed.ncbi.nlm.nih.gov/6377667/
- Lekcharoensuk C, et al. (2001). Association between dietary factors and calcium oxalate and magnesium ammonium phosphate urolithiasis in cats. JAVMA 219(9):1228-37. https://doi.org/10.2460/javma.2001.219.1228